Lijst geblokkeerde donoren 2022-2025

PUBLICATIE LIJST 29 GEBLOKKEERDE DONOREN

Version Français en bas - English version down below

Op dinsdag 18 november 2025, werd in de Kamer tijdens een commissie over de donorproblematiek door het FAGG een lijst van 29 spermadonoren getoond die tussen 2022 en 2025 geblokkeerd werden. Deze donoren werden geblokkeerd omdat er ernstige medische risico’s bij hen of diens donorkinderen vastgesteld werden. In een aantal gevallen lag het overlijden van een donorkind aan de basis van de uiteindelijke blokkering.

Bij alle 29 donoren werd bovendien de wettelijke limiet overschreden: in België mag het sperma van één donor maar gebruikt worden om gezinnen voor maximaal zes vrouwen te vormen. Minister Vandenbroucke vertelde eerder dat het in deze gevallen om 7 tot 27 gezinnen per donor gaat.

Uit goede bron vernamen we dat betrokken ouders en donorkinderen nog niet ingelicht werden over de overschrijding, mogelijks zelfs nog niet over genetische en medische risico's.

Omdat wij vinden dat alle betrokkenen recht hebben op informatie die hem of haar aanbelangt, publiceren we de lijst.

Hoe weet je of jij tot één van deze groepen behoort?

Je bent normaal gezien tussen 2022 en 2025 door je ziekenhuis geïnformeerd dat de donor geblokkeerd werd, met uitleg over de medische reden.
In de lijst hieronder kan je zien wanneer welke donor geblokkeerd werd en waarom.
Komt de medische reden overeen met wat jouw ziekenhuis vertelde? En werd je kort na de blokkering geïnformeerd? Dan is de kans groot dat je tot de betrokken groep behoort.
Hieronder kan je de lijst terugvinden. Je kan hem ook hier downloaden.

Weet dat je ons altijd mag contacteren, daar we zelf al over iets meer informatie beschikken dan hieronder getoond. We kunnen ook helpen in informatie te vergaren. Daarnaast kunnen we je mogelijks met andere gezinnen in contact brengen.

Donor	Bank	Date of block	# families	Descripiton
Donor 1	ESB	23MAY2022	7	Donor is a carrier of classic galactosemia/Shwachman-Diamond syndrome.
Donor 2	ESB	11JUL2022	19	A pathogenic variant in the PHA gene has been identified in donor and he is a carrier of PKU.
Donor 3	ESB	02SEP2022	10	Donor is a carrier of autosomal recessive inherited mild-to-moderate sensorineural hearing loss due to a STRC deletion.
Donor 4	ESB	15DEC2022	14	The donor is a carrier of congenital disorder of glycosylation type Ia.
Donor 5	ESB	18JAN2023	17	Hip dysplasia in a donor confers an increased risk to his offspring.
Donor 6	ESB	20MAR2023	9	Donor is a carrier of c.10955delC(p.Pro3652Glnfs*2) in the PKHD1 gene.
Donor 7	ESB	24MAR2023	11	A deletion of exon 11-14 in the GLI2 gene in a donor child does confer an increased risk to donor’s offspring.
Donor 8	ESB	15JUN2023	8	Donor is compound heterozygous for hemochromatosis.
Donor 9	ESB	04JUL2023	8	A duplication of 22q11 in the donor might confer an increased risk to his offspring.
Donor 10	ESB	05JUL2023	12	Hearing impairment in a donor child may confer an increased risk to donor’s offspring.
Donor 11	ESB	08AUG2023	11	A 57 kb deletion in CTNS-gene in a donor child does confer an increased risk to donor’s offspring.
Donor 12	ESB	30OCT2023	38	TP53 variant in a donor does confer an increased risk to donor’s offspring. Donor number 7069, alias 'Kjeld'.
Donor 13	ESB	08NOV2023	11	Child diagnosed with SCID, severe combined immune defiency. Due to the fact that donor is known carrier of a recessive gene variant.
Donor 14	ESB	07DEC2023	12	Zellweger syndrome in a donor child does confer an increased risk to donor’s offspring.
Donor 15	ESB	20DEC2023	7	Isovaleric acidemia in a donor child, and subsequent genetic analysis showing that donor is heterozygous carrier of a pathogenic variant in the IVD gene; c.158G>A, p.Arg53His, does confer an increased risk to donor’s offspring.
Donor 16	ESB	31JAN2024	10	Hydronephrosis in a donor child does confer an increased risk to donor’s offspring.
Donor 17	ESB	1MAY2024	18	MSH2 variant in a donor does confer an increased risk to donor’s offspring. His gametes are to be permanently blocked.
Donor 18	ESB	25JUN2024	14	A pathogenic variant in the GAA gene does confer an increased risk to a donor child. His gametes are to be permanently blocked.
Donor 19	ESB	07NOV2024	8	Pathogenic MYBPC3 variant in a fetus and found in the donor confers an increased risk to donor’s offspring.
Donor 20	ESB	19NOV2024	17	Cardiomyopathy in a donor child and subsequent diagnosis of heterozygosity of a pathogenic variant in APLK3 gene in the donor confers an increased risk to donor’s offspring. The donor is blocked and his gametes can no longer be used.
Donor 21	ESB	20JAN2025	10	Child was diagnosed with type IV spinal muscular atrophy (SMA). SMA is an autosomal recessive disease. The risk for future donor children is significantly increased.
Donor 22	ESB	21JAN2025	17	Heterozygous deletions at 2p16.3 involving NRXN1 and intragenic mutations of the same gene have been reported in individuals affected by a wide spectrum of neurodevelopmental and psychiatric disorders, including isolated intellectual disability (ID)/global develop-mental delay (GDD), autism spectrum disorder (ASD), attention deficit hyperactivity disorder (ADHD), schizophrenia and bipolar disorder. In a proportion of these patients, neurological involvement (i.e. epilepsy),facial dysmorphism, and congenital heart defects have been observed as associated features. Genotype–phenotype correlations have also been reported.
Donor 23	Cryos	25FEB2025	9	A child has been diagnosed with Usher Syndrome type 2 and genetic test has found two mutations in USH2A. Usher Syndrome type 2 is inherited in a recessive manner, which means that both genetic parents most likely are carriers of the condition. The risk of an affected child is lower than 1%. Donor gametes were blocked for new customers (<1% recurrence risk) 02NOV2022 but can be used for siblings.
Donor 24	ESB	30APR2025	27	A child is reported to have Marfan syndrome. The variant FBN1 (NM_000138.4): c.-5880_164+4097del heterozygot.
Donor 25	ESB	07JUL2025	19	The donor is found to be a healthy carrier of a pathogenic variant in the CYP21A2 gene. Non-classical congenital adrenal hyperplasia (CAH) due to compound heterozygous mutations in the CYP21A2 gene in a donor child confers an increased risk to donor’s offspring.
Donor 26	ESB	19AUG2025	9	Deletion of the CYP21A2 gene in a donor does confer an increased risk to donor’s offspring.
Donor 27	ESB	13OCT2025	12	A pathogenic (class V) genetic variant was observed in the Thyroglobulin (TG) gene. Congenital hypothyroidism in a donor child does confer an increased risk to donor’s offspring.
Donor 28	ESB	20OCT2025	22	The donor is a carrier of Metachromatic leukodystrophy (MLD), since a pathogenic genetic variant in the Arylsulfatase A (ARSA) gene is identified in a heterozygous state. A pathogenic genetic variant :NM_000487.6:c.917C>T, identified in the donor does confer an increased reproductive risk.
Donor 29	ESB	OCT/NOV2025	15	The donor has been under investigation and the results have shown that he is a healthy carrier of Spinal Muscular Atrophy, since MLPA analysis shows a deletion of one of the SMN1 genes.

Wat kan je doen als je tot één van de groepen behoort?

1. Contacteer je fertiliteitscentrum en het FAGG

Vraag hen om:

Bevestiging dat jij tot één van de groepen behoort
De jaren waarin kinderen met hetzelfde sperma werden geboren
Het nummer en alias van de donor (bij de TP53-donor is dat door de minister openbaar gemaakt; jij hebt recht op dezelfde informatie)
Te zeggen tot het hoeveelste gezin jij behoort. Vanaf het 7e gezin heb je meer juridische mogelijkheden richting overheid, FAGG, ziekenhuis en/of spermabank
E-mailadressen FAGG: icm@fagg.be en management@fagg.be.

2. Overweeg om een klacht in te dienen bij de politie of het parket

Zo kan er een onafhankelijk onderzoek gestart worden. Dat is belangrijk om tijdig duidelijkheid te krijgen over mogelijke fouten en aansprakelijkheid.

3. Neem contact op met een advocaat

Heb je een rechtsbijstandsverzekering? Dan worden de kosten meestal gedekt.
Een rechtszaak kan helpen om schadevergoeding te vragen en verantwoordelijkheden af te dwingen.
Niet alleen gezinnen 7 en hoger kunnen stappen ondernemen; ook het 1ste tot het 6e gevormde gezin kunnen dit overwegen. Zij lijden namelijk ook schade en gevolgen door dat wetgeving en contracten niet nageleefd werden.

4. Contacteer ons

Wij kunnen helpen door:

Informatie te verzamelen of door te geven.
Je in contact te brengen met andere betrokken ouders of donorkinderen.

LISTE PUBLIÉE DES 29 DONNEURS BLOQUÉ

English version down below

Le mardi 18 novembre 2025, lors d’une commission à la Chambre concernant la problématique des donneurs, l’AFMPS a présenté une liste de 29 donneurs de sperme ayant été bloqués entre 2022 et 2025. Ces donneurs ont été bloqués en raison de risques médicaux graves identifiés chez eux ou chez leurs enfants issus du don. Dans plusieurs cas, le décès d’un enfant issu du don a mené à la décision finale de les bloquer.

Pour les 29donneurs, la limite légale a en outre été dépassée : en Belgique, le sperme d’un même donneur ne peut être utilisé que pour former des familles pour un maximum de six femmes. Le ministre Vandenbroucke avait déjà indiqué que, dans ces cas, il s’agissait de 7 à 27 familles par donneur.

D’après nos informations, les parents concernés et les personnes issues du don n’ont pas encore été informés du dépassement de cette limite, et peut-être même pas des risques génétiques ou médicaux.

Parce que nous estimons que toutes les personnes concernées ont droit aux informations qui les touchent directement, nous publions cette liste

Comment savoir si vous faites partie des groupes concernés ?

Normalement, entre 2022 et 2025, votre hôpital vous a informé que le donneur avait été bloqué et vous a expliqué la raison médicale.
Dans la liste ci-dessous, vous pouvez voir quand chaque donneur a été bloqué et pour quelle raison.
La raison médicale correspond-elle à ce que votre hôpital vous a communiqué ? Et avez-vous été informé(e) peu après le blocage ? Alors il est probable que vous fassiez partie du groupe concerné.
Vous trouverez la liste ci-dessous. Vous pouvez aussi la télécharger ici.

Sachez que vous pouvez toujours nous contacter : nous disposons déjà de quelques informations supplémentaires par rapport à ce qui est présenté ci-dessous. Nous pouvons également vous aider à rassembler des informations et éventuellement vous mettre en contact avec d’autres familles.

Donor	Bank	Date of block	Descripiton
Donor 1	ESB	23MAY2022	Donor is a carrier of classic galactosemia/Shwachman-Diamond syndrome.
Donor 2	ESB	11JUL2022	A pathogenic variant in the PHA gene has been identified in donor and he is a carrier of PKU.
Donor 3	ESB	02SEP2022	Donor is a carrier of autosomal recessive inherited mild-to-moderate sensorineural hearing loss due to a STRC deletion.
Donor 4	ESB	15DEC2022	The donor is a carrier of congenital disorder of glycosylation type Ia.
Donor 5	ESB	18JAN2023	Hip dysplasia in a donor confers an increased risk to his offspring.
Donor 6	ESB	20MAR2023	Donor is a carrier of c.10955delC(p.Pro3652Glnfs*2) in the PKHD1 gene.
Donor 7	ESB	24MAR2023	A deletion of exon 11-14 in the GLI2 gene in a donor child does confer an increased risk to donor’s offspring.
Donor 8	ESB	15JUN2023	Donor is compound heterozygous for hemochromatosis.
Donor 9	ESB	04JUL2023	A duplication of 22q11 in the donor might confer an increased risk to his offspring.
Donor 10	ESB	05JUL2023	Hearing impairment in a donor child may confer an increased risk to donor’s offspring.
Donor 11	ESB	08AUG2023	A 57 kb deletion in CTNS-gene in a donor child does confer an increased risk to donor’s offspring.
Donor 12	ESB	30OCT2023	TP53 variant in a donor does confer an increased risk to donor’s offspring. Donor number 7069, alias 'Kjeld'.
Donor 13	ESB	08NOV2023	Child diagnosed with SCID, severe combined immune defiency. Due to the fact that donor is known carrier of a recessive gene variant.
Donor 14	ESB	07DEC2023	Zellweger syndrome in a donor child does confer an increased risk to donor’s offspring.
Donor 15	ESB	20DEC2023	Isovaleric acidemia in a donor child, and subsequent genetic analysis showing that donor is heterozygous carrier of a pathogenic variant in the IVD gene; c.158G>A, p.Arg53His, does confer an increased risk to donor’s offspring.
Donor 16	ESB	31JAN2024	Hydronephrosis in a donor child does confer an increased risk to donor’s offspring.
Donor 17	ESB	1MAY2024	MSH2 variant in a donor does confer an increased risk to donor’s offspring. His gametes are to be permanently blocked.
Donor 18	ESB	25JUN2024	A pathogenic variant in the GAA gene does confer an increased risk to a donor child. His gametes are to be permanently blocked.
Donor 19	ESB	07NOV2024	Pathogenic MYBPC3 variant in a fetus and found in the donor confers an increased risk to donor’s offspring.
Donor 20	ESB	19NOV2024	Cardiomyopathy in a donor child and subsequent diagnosis of heterozygosity of a pathogenic variant in APLK3 gene in the donor confers an increased risk to donor’s offspring. The donor is blocked and his gametes can no longer be used.
Donor 21	ESB	20JAN2025	Child was diagnosed with type IV spinal muscular atrophy (SMA). SMA is an autosomal recessive disease. The risk for future donor children is significantly increased.
Donor 22	ESB	21JAN2025	Heterozygous deletions at 2p16.3 involving NRXN1 and intragenic mutations of the same gene have been reported in individuals affected by a wide spectrum of neurodevelopmental and psychiatric disorders, including isolated intellectual disability (ID)/global develop-mental delay (GDD), autism spectrum disorder (ASD), attention deficit hyperactivity disorder (ADHD), schizophrenia and bipolar disorder. In a proportion of these patients, neurological involvement (i.e. epilepsy),facial dysmorphism, and congenital heart defects have been observed as associated features. Genotype–phenotype correlations have also been reported.
Donor 23	Cryos	25FEB2025	A child has been diagnosed with Usher Syndrome type 2 and genetic test has found two mutations in USH2A. Usher Syndrome type 2 is inherited in a recessive manner, which means that both genetic parents most likely are carriers of the condition. The risk of an affected child is lower than 1%. Donor gametes were blocked for new customers (<1% recurrence risk) 02NOV2022 but can be used for siblings.
Donor 24	ESB	30APR2025	A child is reported to have Marfan syndrome. The variant FBN1 (NM_000138.4): c.-5880_164+4097del heterozygot.
Donor 25	ESB	07JUL2025	The donor is found to be a healthy carrier of a pathogenic variant in the CYP21A2 gene. Non-classical congenital adrenal hyperplasia (CAH) due to compound heterozygous mutations in the CYP21A2 gene in a donor child confers an increased risk to donor’s offspring.
Donor 26	ESB	19AUG2025	Deletion of the CYP21A2 gene in a donor does confer an increased risk to donor’s offspring.
Donor 27	ESB	13OCT2025	A pathogenic (class V) genetic variant was observed in the Thyroglobulin (TG) gene. Congenital hypothyroidism in a donor child does confer an increased risk to donor’s offspring.
Donor 28	ESB	20OCT2025	The donor is a carrier of Metachromatic leukodystrophy (MLD), since a pathogenic genetic variant in the Arylsulfatase A (ARSA) gene is identified in a heterozygous state. A pathogenic genetic variant :NM_000487.6:c.917C>T, identified in the donor does confer an increased reproductive risk.

Que pouvez-vous faire si vous faites partie de l’un des groupes concernés ?

1. Contactez votre centre de fertilité et l’AFMPS

Demandez-leur :

Adresses e-mail de l’AFMPS : icm@fagg.be en management@fagg.be.

- La confirmation que vous faites bien partie de l’un des groupes concernés
- Les années durant lesquelles des enfants sont nés avec le même sperme
- Le numéro et l’alias du donneur (pour le donneur porteur de TP53, ces informations ont été rendues publiques par le ministre ; vous avez droit aux mêmes renseignements)
- De vous indiquer à quelle famille vous correspondez. À partir de la 7ᵉ famille, vous disposez de davantage de possibilités juridiques envers les autorités, l’AFMPS, l’hôpital et/ou la banque de sperme

2. Envisagez de déposer une plainte auprès de la police ou du parquet

Cela peut permettre l’ouverture d’une enquête indépendante. C’est essentiel pour obtenir rapidement des éclaircissements sur d’éventuelles erreurs et sur les responsabilités.

3. Contactez un avocat

Disposez-vous d’une assurance protection juridique ? Dans ce cas, les frais sont généralement couverts.
Une action en justice peut vous aider à demander une indemnisation et à faire reconnaître les responsabilités.
Non seulement les familles 7 et suivantes peuvent entreprendre des démarches, mais les familles 1 à 6 peuvent également le faire, car elles subissent elles aussi un préjudice et des conséquences du fait que la législation et les contrats n’ont pas été respectés.

4. Contactez-nous
Nous pouvons vous aider en :

Rassemblant ou transmettant des informations
Vous mettant en contact avec d’autres parents concernés ou avec des personnes issues du don

PUBLICATION OF THE LIST OF 29 BLOCKED SPERM DONORS

On Tuesday, 18 November 2025, during a parliamentary committee meeting on donor-related issues, the FAMHP presented a list of 29 sperm donors who were blocked between 2022 and 2025. These donors were blocked because serious medical risks were identified in them or in their donor-conceived children. In several cases, the death of a donor-conceived child was the reason that ultimately led to the blocking.

For all 29 donors, the legal limit was exceeded: in Belgium, the sperm of one donor may only be used to create families for a maximum of six women. Minister Vandenbroucke previously stated that in these cases, each donor was linked to between 7 and 27 families.

Reliable sources have informed us that the parents and donor-conceived individuals concerned have not yet been informed about this exceeding of the limit, and possibly not even about the genetic and medical risks.

Because we believe that every person involved has the right to information that concerns them, we are publishing the list.

How do you know if you belong to one of these groups?

Normally, your hospital should have informed you between 2022 and 2025 that the donor had been blocked, along with an explanation of the medical reason.
In the list below, you can see when each donor was blocked and for what reason.
Does the medical reason match what your hospital told you? And were you informed shortly after the blocking? Then it is likely that you belong to the group concerned.
You can find the list below. You can also download it here.

Please know that you can always contact us, as we already have access to slightly more information than what is shown below. We can also help you gather information. Additionally, we may be able to put you in touch with other families.

Donor	Bank	Date of block	Descripiton
Donor 1	ESB	23MAY2022	Donor is a carrier of classic galactosemia/Shwachman-Diamond syndrome.
Donor 2	ESB	11JUL2022	A pathogenic variant in the PHA gene has been identified in donor and he is a carrier of PKU.
Donor 3	ESB	02SEP2022	Donor is a carrier of autosomal recessive inherited mild-to-moderate sensorineural hearing loss due to a STRC deletion.
Donor 4	ESB	15DEC2022	The donor is a carrier of congenital disorder of glycosylation type Ia.
Donor 5	ESB	18JAN2023	Hip dysplasia in a donor confers an increased risk to his offspring.
Donor 6	ESB	20MAR2023	Donor is a carrier of c.10955delC(p.Pro3652Glnfs*2) in the PKHD1 gene.
Donor 7	ESB	24MAR2023	A deletion of exon 11-14 in the GLI2 gene in a donor child does confer an increased risk to donor’s offspring.
Donor 8	ESB	15JUN2023	Donor is compound heterozygous for hemochromatosis.
Donor 9	ESB	04JUL2023	A duplication of 22q11 in the donor might confer an increased risk to his offspring.
Donor 10	ESB	05JUL2023	Hearing impairment in a donor child may confer an increased risk to donor’s offspring.
Donor 11	ESB	08AUG2023	A 57 kb deletion in CTNS-gene in a donor child does confer an increased risk to donor’s offspring.
Donor 12	ESB	30OCT2023	TP53 variant in a donor does confer an increased risk to donor’s offspring. Donor number 7069, alias 'Kjeld'.
Donor 13	ESB	08NOV2023	Child diagnosed with SCID, severe combined immune defiency. Due to the fact that donor is known carrier of a recessive gene variant.
Donor 14	ESB	07DEC2023	Zellweger syndrome in a donor child does confer an increased risk to donor’s offspring.
Donor 15	ESB	20DEC2023	Isovaleric acidemia in a donor child, and subsequent genetic analysis showing that donor is heterozygous carrier of a pathogenic variant in the IVD gene; c.158G>A, p.Arg53His, does confer an increased risk to donor’s offspring.
Donor 16	ESB	31JAN2024	Hydronephrosis in a donor child does confer an increased risk to donor’s offspring.
Donor 17	ESB	1MAY2024	MSH2 variant in a donor does confer an increased risk to donor’s offspring. His gametes are to be permanently blocked.
Donor 18	ESB	25JUN2024	A pathogenic variant in the GAA gene does confer an increased risk to a donor child. His gametes are to be permanently blocked.
Donor 19	ESB	07NOV2024	Pathogenic MYBPC3 variant in a fetus and found in the donor confers an increased risk to donor’s offspring.
Donor 20	ESB	19NOV2024	Cardiomyopathy in a donor child and subsequent diagnosis of heterozygosity of a pathogenic variant in APLK3 gene in the donor confers an increased risk to donor’s offspring. The donor is blocked and his gametes can no longer be used.
Donor 21	ESB	20JAN2025	Child was diagnosed with type IV spinal muscular atrophy (SMA). SMA is an autosomal recessive disease. The risk for future donor children is significantly increased.
Donor 22	ESB	21JAN2025	Heterozygous deletions at 2p16.3 involving NRXN1 and intragenic mutations of the same gene have been reported in individuals affected by a wide spectrum of neurodevelopmental and psychiatric disorders, including isolated intellectual disability (ID)/global develop-mental delay (GDD), autism spectrum disorder (ASD), attention deficit hyperactivity disorder (ADHD), schizophrenia and bipolar disorder. In a proportion of these patients, neurological involvement (i.e. epilepsy),facial dysmorphism, and congenital heart defects have been observed as associated features. Genotype–phenotype correlations have also been reported.
Donor 23	Cryos	25FEB2025	A child has been diagnosed with Usher Syndrome type 2 and genetic test has found two mutations in USH2A. Usher Syndrome type 2 is inherited in a recessive manner, which means that both genetic parents most likely are carriers of the condition. The risk of an affected child is lower than 1%. Donor gametes were blocked for new customers (<1% recurrence risk) 02NOV2022 but can be used for siblings.
Donor 24	ESB	30APR2025	A child is reported to have Marfan syndrome. The variant FBN1 (NM_000138.4): c.-5880_164+4097del heterozygot.
Donor 25	ESB	07JUL2025	The donor is found to be a healthy carrier of a pathogenic variant in the CYP21A2 gene. Non-classical congenital adrenal hyperplasia (CAH) due to compound heterozygous mutations in the CYP21A2 gene in a donor child confers an increased risk to donor’s offspring.
Donor 26	ESB	19AUG2025	Deletion of the CYP21A2 gene in a donor does confer an increased risk to donor’s offspring.
Donor 27	ESB	13OCT2025	A pathogenic (class V) genetic variant was observed in the Thyroglobulin (TG) gene. Congenital hypothyroidism in a donor child does confer an increased risk to donor’s offspring.
Donor 28	ESB	20OCT2025	The donor is a carrier of Metachromatic leukodystrophy (MLD), since a pathogenic genetic variant in the Arylsulfatase A (ARSA) gene is identified in a heterozygous state. A pathogenic genetic variant :NM_000487.6:c.917C>T, identified in the donor does confer an increased reproductive risk.

What can you do if you belong to one of the groups?

1. Contact your fertility centre and the FAMHP

Ask them for:

Confirmation that you are part of one of the affected groups
The years in which children were born using the same sperm
The donor’s number and alias (for the TP53 donor, this was made public by the minister; you are entitled to the same information)
Information on which family number you are. From the 7th family onward, you have more legal options towards the government, the FAMHP, the hospital and/or the sperm bank

FAMHP email addresses: icm@fagg.be en management@fagg.be.

2. Consider filing a complaint with the police or the public prosecutor’s office

This can initiate an independent investigation. It is important to obtain timely clarity about possible errors and liability.

3. Contact a lawyer

Do you have legal assistance insurance? If so, the costs are usually covered.
A legal action can help you seek compensation and enforce accountability.
Not only families 7 and above can take action; families 1 to 6 may also consider doing so, as they too suffer harm and consequences from the failure to respect legislation and contractual obligations.

4. Contact us

We can help by: